People who’ve heard of hip dysplasia often think of it in connection with babies and dogs. One baby in 1,000 is born with hip dysplasia, but only 12 percent of those have unstable hips past the age of 2 months [source: Ramsey]. And hip dysplasia is common in dogs, particularly in large breeds. Hip dysplasia doesn’t occur only in infants and pets, though. People, particularly women, can be diagnosed with and treated for hip dysplasia as adults.
Hip dysplasia is an abnormal condition affecting the hip socket, or acetabulum, and the thighbone, or femur. In someone with hip dysplasia, the hip socket is too shallow, or the femur is in the wrong place. Historically, many doctors have referred to hip dysplasia as congenital dysplasia of the hip (CDH). However, in the last decade or so, developmental dysplasia of the hip (DDH) has become the accepted term, since hip dysplasia can either exist from birth or develop in the first few weeks, months or years of life [source: Larson et al]. Eighty percent of all people with DDH are female [source: Ramsey].
Doctors check for hip dysplasia in babies before sending them home from the hospital — it’s an easily treatable condition in newborns. But if hip dysplasia isn’t detected and corrected at an early age, the health of the hip will deteriorate over time because of its misalignment. Depending on the severity of the situation, this could result in pain, osteoarthritis, difficulty walking or an inability to walk. It can also contribute to a marked deterioration in a person’s basic quality of life.
How can a baby’s development lead to a hip socket that’s not deep enough, and what can doctors do to treat the issue? If someone discovers she has hip dysplasia as an adult, what are her options for treatment?
Hip Dysplasia and Hip Anatomy
To understand how hip dysplasia occurs and how doctors treat it, you need to know a little bit about the hip joint itself. The hip is a ball-in-socket joint, or an enarthrosis. The rounded head of the femur forms the ball, which fits into the socket of the acetabulum. The hip forms the primary connection between the bones of the lower limbs and the axial skeleton of the trunk and pelvis.
Three pelvic bones — the ilium, the ischium and the pubis — come together to form the acetabulum. The joint itself may not be fully ossified, or hardened into bone, until a person reaches the age of 25 years, which is one reason why early detection is important in hip dysplasia treatment. A strong, lubricated layer of articular hyaline cartilage covers both surfaces of the joint completely, helping it move more smoothly. A rim called the labrum grips the head of the femur and secures it in the joint. This increases the depth of the acetabulum.
While in the uterus, a baby’s hip should develop with the femoral head sitting perfectly centered in the acetabulum. The acetabulum should cover the head of the femur as if it were a ball sitting inside of a cup. In congenital hip dysplasia, the development of the acetabulum allows the femoral head to ride upward out of the socket, especially when the baby begins to walk. This results in a shallow socket — it’s shaped less like a cup and more like a bowl. It can also lead to femoral misplacement because the acetabulum doesn’t cover the femoral head sufficiently.
If this isn’t corrected, the hip joint will be unstable. In some cases, dislocation may also occur. Over the years, as the femoral head and acetabulum move without correct alignment, the cartilage in the joint wears down prematurely and unevenly. The result is differing degrees of osteoarthritis, depending on the severity of the misalignment. The osteoarthritis can occur on the joint head or in the socket itself.
The exact anatomical features of hip dysplasia can vary from person to person. Both the hip socket and the femur can be affected.
An atypically shaped thighbone can also be part of hip dysplasia. This can occur in addition to or in place of a misshaped acetabulum. The ball and shaft of the femur ideally form a 120- to 135-degree angle where they meet. But in hip dysplasia, that angle can change in one of two ways:
- Coxa valga: The angle between the ball and the shaft of the femur is increased, usually above 135 degrees.
- Coxa vara: The angle is reduced to less than 120 degrees.
In either case, the length of the femur is affected — it’s shorted with coxa vara or lengthened with coxa valga. This creates additional imbalance within the hip joint and results in difficulty walking, pain and joint stiffness.
Doctors don’t know exactly what causes hip dysplasia. However, a possible cause could be hormonal changes within the mother. During pregnancy, hormones loosen a woman’s joints to allow them to expand and make room for the baby during delivery. These hormones are thought to cross over the placenta and cause the baby to have increased ligament looseness. The baby’s position in the womb may play a role as well — breech birth positioning and firstborn children have increased rates of developmental dysplasia of the hip (DDH). As many as one in 15 females born in breech position has DDH [source: Ramsey]. The left leg is more often involved due to the baby’s positioning in the uterus. There is also a 10-fold increase in the frequency of hip dysplasia in children whose parents had DDH compared with those whose parents did not [source: Bjerkreim].